Pssc Research Paper

Hypertrophic cardiomyopathy is an inherited disease that affects the cardiac muscle of the heart, causing the walls of the heart to thicken and become stiff. [1] On a cellular level, the sarcomere

The SAN policy controls whether a newly discovered disk is brought online or stays offline, and whether it turns out to be read/write or if it remains read-only.

According to the WHO, cardiovascular diseases have been the leading cause of death globally claiming 17 million lives a year, more deaths than all cancer combined (Chiu and Radisic, 2013). Cardiovascular disease is responsible for a preponderance of health problems and its impact is expected to grow further as the population ages. In the UK, NHS spends about £7.74 billion as the expenditure to deal with cardiovascular diseases (Barton et al., 2011). Cardiovascular disease in the form of myocardial infarction has become the principle cause of death in developed countries, accounting for nearly 40% of all deaths (http://www.bhf.org.uk/). Congenital heart defects, which occur in nearly 14 of every 1000 new-born children, is another tragic fact that baffles medical industry (http://www.heart.org/). About 61 million Americans (almost one-fourth of the population) live with cardiovascular diseases, such as coronary heart disease, congenital cardiovascular defects, and congestive heart failure.

The years go by to fast so choosing the colleges or universities is very important in our juniors years and senior years. It's better to be ready for college and have your future set go. Katy Hopkins “there more than one college you can go to” which is very true. There are a lot waiting and you can go too. Setting the pros and cons are very important to set for college, Katy Hopkins says “rank your priorities”. The college can help you with loans, Katy Hopkins says to “compare financial aid pages” which is right to find out the better college to help. Staying close to the college is very smart to do, safe gas and money Katy Hopkins says “go back to school” which is saying to stay close to it as you can.

Critical Congenital Heart Defects are abnormalities of the heart structure that are present at birth. These occur because of incomplete or abnormal development of the fetus’ heart. These defects can cause severe mortality within the newborn stage (Goldstein, 2013, p.1). Several are known to be linked to genetic disorders such as Down syndrome and others are thought to be linked to environmental factors that women can be exposed to while pregnant. The cause of most CCHD’s is unknown.

Cardiomyopathy is a disease of the heart muscle. It is where the ventricular structure is impaired. The heart muscle

Presidio is wonderful Place to live. Presidio maybe Hot in the summer. Summer Here in Presidio you can do as many things you want To Do. Presidio Is a Wonderful place to live In the Summer. You can go to the Awesome Presidio pool. The pool in presidio is refreshing, Cold and awesome. Presidio is A Wonderful Place to live In the Summer. You get to do Really Fun things. Presidio Is Sometimes hot But If You Like To Stay Inside And play basketball you are in luck because here in presidio, we have an activity center with an indoor basketball court and you can play in the PAC center free. The PAC center is cool and has large A/C vents. In Presidio, it is hot but some people do not go outside. I go outside in the summer so I can play baseball on the

TOF is a CHD which means the abnormal development of the heart occurred during gestation .A normal heart consists of four chambers of cardiac muscle cells, four heart valves, an electrical pathway, and its own blood supply. The chambers are the Right Atrium (RA) , Right Ventricle ( RV), Left Atrium,(LA) , and the Left Ventricle ( LV). The blood flows from the Vena Cava to the RA thru the Tricuspid Valve to the RV. It then gets ejected thru the Pulmonary Valve into the Pulmonary Artery, and on into the lungs. It returns to the LA, goes thru the Mitral Valve into the LV, and then is ejected thru the Aortic Valve into the aorta and onto systemic circulation. TOF is identified by four atypical finding within the heart.

Hypertrophic cardiomyopathy (HCM) is a primary disease of the myocardium (the muscle of the heart) in which a portion of the myocardium ishypertrophied (thickened) without any obvious cause. It is perhaps best known as a leading cause of sudden cardiac death in young athletes.[7] The occurrence of hypertrophic cardiomyopathy is a significant cause of sudden unexpected cardiac death in any age group and as a cause of disabling cardiac symptoms. Younger people are likely to have a more severe form of hypertrophic cardiomyopathy.

These separate forms of the condition come with distinct traits that vary from one to the next. Dilated cardiomyopathy, “The most common form, this occurs when the muscle fibers of one or both pumping chambers are weak and unable to contract normally” (“Understand Pediatric” 1). Which means that the heart beats abnormally due to lack of blood flow. Often with DCM the heart is weak and fails because it cannot sufficiently pump blood throughout the body (“Understanding Pediatric” 1). The article on “Understanding Pediatric Cardiomyopathy” states that hypertrophic cardiomyopathy is the second most common form. HCM has a few characteristics that can be mistaken for dilated cardiomyopathy. Thick and stiff muscles may stop the heart from contracting properly, which may narrow the passageways for blood to move out of the heart (“Understanding Pediatric” 1). So, the muscles within the heart thicken to try and help blood flow. In an advanced form of hypertrophic cardiomyopathy, there is a chance that the muscles may thin to try to help the poor pumping action (“Understanding Pediatric” 1). When HCM tries to help the body it, in itself, can cause failure. A

Hypertrophic cardiomyopathy is a genetic disease that causes the heart muscle to become thickened (Maron, 2015). According to the Mayo Clinic (n.d.), hypertrophic cardiomyopathy is a genetic disorder; however, environmental or lifestyle factors may influence how and when the disease appears in individuals.

Pathological cardiac hypertrophy is associated structural remodeling which can include cardiomyocyte growth, fibrosis (formation of collagen fibril connective tissue) and angiogenesis (formation of new blood vessels).

Hypertrophic Cardiomyopathy is also known as HCM. It is a genetic condition in which the myocardium or heart muscle in the left ventricle is thickened. HCM cannot be identified by a single complaint or symptom as it is different for every person that it affects. This disease not only causes left ventricle hypertrophy but can also cause ventricle stiffness, mitral valve changes, and other cellular changes.

Metabolic syndrome is a group of disorders that predisposes to the cardiovascular disorders and constituted a significant risk for coronary heart disease, however, its effect on gene expression pattern is unclear. The considerable evidence demonstrated that genetic factors predispose to the cardiovascular complications. Metabolic syndrome significantly alters cardiac gene expression profile which may be involved in the development of cardiovascular disease.

Arrhythmogenic right ventricular cardiomyopathy (ARVC) (1) is an inherited disease characterized by the progressive replacement of the myocardium by adipose and fibrous tissue (2) that predisposes to the development of ventricular tachycardia (VT) and to sudden cardiac death (SCD). This condition was described three decades ago, when fibro-fatty infiltration in the right ventricle was considered its pivotal indicator (3,4). It later became clear that the ARVC is mainly caused by mutations in the genes encoding for desmosomal proteins (1). This helped unveil that the disorder is often associated with biventricular manifestations (5) and the term "arrhythmogenic cardiomyopathy" has also been proposed (1). The unmet need in the management