Parkinson Research Paper

James Parkinson first discovered Parkinson's Disease in 1817. Parkinson's Disease is a common neurologic disorder for the elderly. It is a disorder of the brain characterized by shaking and difficulty with walking, movement, and coordination. This disease is associated with damage to a part of the brain that controls muscle movement. Parkinson's Disease is a chronic illness that is still being extensively studied.

Diagnosis of Parkinson’s Disease is extremely important in terms of treating the symptoms before the disease gets worse. It is common for patients with PD to have motor symptoms such as gait disorder, which comes from muscle stiffness/rigidity, bradykinesia, postural imbalance, etc. Gait disorders can generally help determine how far the neurological disorder has affected the motor function and control of the individual. Many physicians in general clinics determine if a patient has PD or if it has progressed through evaluating the patients gait pattern, focused on a direct path walking. The problem with this way of diagnosis is that direct paths would generally require the clinic to have a space of 100-meter length for the patient to walk;

Parkinson disease (PD) is one of the most common neurologic disorders. and it affects approximately 1% of individuals older than 60 years old. Parkinson’s disease is a condition that progresses slowly by treatment. In addition, loss of pigmented dopaminergic neurons of the substantianigra pars compacta and the presence of Lewy bodies and Lewyneurites are the two major neuropathologic findings in Parkinson disease (Hauser, 2016).

Parkinson’s disease is a “neurodegenerative disorder of the basal nuclei due to insufficient secretion of the neurotransmitter dopamine” (Marieb & Hoehn, 2013, p. G-17). The cause of Parkinson’s disease is unknown, but many factors play a role in the development of Parkinson’s disease. One factor that has been found in an individual who has Parkinson’s disease causes over activity of targeted dopamine-deprived basal nuclei. This over activity is caused by the breakdown of neurons that release dopamine in the substantia nigra (Marieb & Hoehn, 2013). Another factor that is present in a person who has Parkinson’s disease, is the presence of lewy bodies in the brain stem ("What is lbd?," 2014). Lewy bodies are unusual

While attending the Parkinson’s Support Group, I observed and took note of many occurrences throughout the group. There were 18 caregivers/individuals with Parkinson’s in attendance, along with 3 students, the director, and the group leader (a counseling student intern). Each individual diagnosed with Parkinson’s, except for two, was accompanied by their spouse/caregiver. Members of this group sat around a rectangle table with the group leader at the front. The purpose of the group, on that particular day, was to discuss challenges that individuals with Parkinson’s and or caregivers face, activities and ways to cope, and some positive gains after the disability. The type of group that was ran was a support group, as compared to having guest speakers as usual. The Parkinson’s group was open to the

Parkinson disease (PD), also referred to as Parkinson’s disease and paralysis agitans, is a progressive neurodegenerative disease that is the third most common neurologic disorder of older adults. It is a debilitating disease affecting motor ability and is characterized by four cardinal symptoms: tremor rigidity, bradykinesia or kinesis (slow movement/no movement), and postural instability. Most people have primary, or idiopathic, disease. A few patients have secondary parkinsonian symptoms from conditions such as brain tumors and certain anti-psychotic drugs.

There is also no actual known cause of the disease. But in your case because of the car accident that you had a few weeks ago The MRI that we took of your brain shows that your brain was hit pretty hard and the nerve cells of your basal ganglia have become impaired which means you will start to produce a lot less dopamine. An MRI is a painless test used to see the inside of the body without using X-rays. It uses a large magnet, safe, low- energy radio waves and a computer to produce 2d or 3d pictures. Radio waves are passed into your body and are absorbed by some of the tissues, which in turn retransmit the radio waves. The magnet is then turned on and off. The computer picks up this information and generates a picture. Diseased tissue gives off a different signal from healthy tissue and the machine detects this. The positives of having an MRI is that it provides a detailed picture of any part of the body and also means that some of the less pleasant tests do not have to be done. The negatives of having an MRI are very little, they include, being slightly uncomfortable if you are claustrophobic, otherwise the test should have gone very smoothly. Also because you are over the age of 50 this can come in role of the disease. The damage of your brain and your age a very high risks of developing Parkinson’s. Through our research, our understanding the possible causes of Parkinson’s disease is increasing all the time. We also know that you have Parkinson’s because of the symptoms you have been showing. In This disease there are four categories- tremors, stiffness, slowness of movement, and impaired balance and coordination. The nonmotor symptoms, which are symptoms that are not visible are- memory loss, depression, diminished sense of smell. All of these symptoms develop slowly and gradually progress over time. Also remember that each person is very different and are affected differently and

Parkinson’s is a progressive neurodegenerative disease, primarily affecting voluntary, precise, and controlled movement. Parkinson’s occurs when cells in a part of the brain called the substantia nigra die off. These cells are responsible for producing dopamine. With less and less dopamine, a person has less and less ability to regulate their movements, body and emotions. The terms "familial Parkinson's disease" and "sporadic Parkinson's disease" are used to differentiate genetic from truly idiopathic forms of the disease.

Parkinson Disease (PD) is a neurodegenerative disease symptomized by tremor, muscular rigidity, and slow imprecise movements. Typically, the disease affects middle-aged and elderly individuals. PD is associated with degeneration of the basal ganglia of the brain causing a deficiency of the neurotransmission of dopamine.

disease” (Atchison & Dirette, 2012, p. 213). The disease is a complex hypokinetic type with

As a young person I was captivated and intrigued by the tales of early explorers and naturalists, especially the British. Captain James Cook’s voyages especially sparked my sense of adventure and curiosity. Being among the first westerners to have contact with unknown cultures must have been magnificent. To be exposed to foreign languages, material culture, flora and fauna and then to have the opportunity to record the life-style and environment for generations to glimpse is extraordinary. Sydney Parkinson was one of these fortunate people who was in the right place -at the right time, to work in the capacity of one of botanical artist on-board the first voyage to the South Seas on board the Endeavor , captained by James Cook and hired by Sir Joseph Banks. He was one of the early explorers to

Parkinson’s Disease is known as one of the most common progressive and chronic neurodegenerative disorders. It belongs to a group of conditions known as movement disorders. Parkinson disease is a component of hypokinetic disorder because it causes a decreased in bodily movement. It affects people who are usually over the age of 50. It can impair an individual motor as well as non-motor function. Some of the primary symptoms of Parkinson’s disease are characterized by tremors or trembling in hands, legs and arms. In early symptoms the tremor can be unilateral, appearing in one side of body but progression in the disease can cause it to spread to both sides; rigidity or a resistant to movement affects most people with Parkinson’s disease,

Parkinson’s Disease-like pathologies are found in different organisms. However, the pathology do not necessarily result in Parkinson’s-like symptoms. In horses, dysfunction of the pituitary pars intermedia due to the loss of dopaminergic neurons within the hypothalamus is a characteristic of Cushing’s disease (Spelta, 2015). Currently, it is hypothesized that with age, oxidation within the hypothalamus causes the loss of dopaminergic neurons. In equine Cushing’s disease, proopiomelanocortin peptides (POMC), such as acetylcholine (ACTH), α-melanocyte-stimulation hormone (α-MSH), and ß-endorphin, are mass-produced due to the lack of dopamine inhibiting melanotrope cells of the pars intermedia within the pituitary gland (Toribio, 2012). With

Parkinson’s Disease is a very common disorder these days. Over 10 million people live daily with Parkinson worldwide. Parkinson’s Disease was named after an English surgeon James Parkinson who wrote a detailed description essay called Shaking Palsy in 1817. The average age for Parkinson’s Disease is between 45 to 70 years old but you can also have juvenile or young onset as well. Most common symptoms of Parkinson are tremors, bradykinesia or akinesia, or rigidity or stiffness, and balance disorder. Parkinson’s Disease doesn’t have a cure and the cause is unknown it could be a number of things genetics, environmental triggers, age, or gender. Parkinson’s Disease happens because the dopaminergic neuron dies and

Parkinson disease (PD) is a progressive neurodegenerative disorder characterized mainly by physical and psychological disabilities. This disorder was named after James Parkinson, an English physician who first described it as shaking palsy in 1817 (Goetz, Factr, and Weiner, 2002). Jean- Martin Charcot, who was a French neurologist, then progressed and further refined the description of the disease and identified other clinical features of PD (Goetz, Factr, and Weiner, 2002). PD involves the loss of cells that produce the neurotransmitter dopamine in a part of the brain stem called the substansia nigra, which results in several signs and symptoms (Byrd, Marks, and Starr, 2000). It is manifested clinically by tremor,